Polycystic Kidney Disease Can Arouse Nephrotic Hypertension

The occurrence of Nephrotic Hypertension caused by Polycystic Kidney Disease (PKD) is resulting from that cysts press normal kidney tissue and ischemia, anoxia and low blood perfusion occur in kidney. Progressive pathological lesion of kidney will happen if PKD patients with Hypertension can not control the disease in time. Renal fibrosis becomes more severe and serum creatinine increases. Aggravating Kidney Disease will further hike blood pressure continuously, which can enlarge heart and cause heart failure. Some patients’ conditions get worse rapidly. Renal Insufficiency may happen if renal tissues are pressed and renal functions are damaged, which will develop into Uremia finally.
Treatment of Nephrotic Hypertension caused by Polycystic Kidney Disease (PKD)
There are some differences between Primary Hypertension and Nephrotic Hypertension caused by Polycystic Kidney Disease (PKD). For the latter, the crux of treatment is to treat Polycystic Kidney Disease (PKD). The priority is to reduce cysts’pressure to kidney and renal parenchyma, postpone or block the progression of Renal Fibrosis. Only by this, can Hypertension be treated radically. The aim of Micro Chinese Medicines is to decrease cysts and reduce the damage of renal tissue fundamentally.

Dilating blood vessels, general blood circulation can be improved so as to attain treatment.

By expanding renal arteries, blood perfusion increases, so do general microcirculations, oxygen supply and metabolism gets improved, which can change the activity of cyst wall cell, increase permeability and bring away cyst liquid. Meanwhile the secretion of cyst wall cell can be blocked by expanding renal arteries, so as to make cysts shrink.


With the active ingredient in Chinese medicine playing efficacy, the pressure of cysts to nephrons around can be relieved; ischemia and anoxia of normal kidney tissue can be moderated so as to delay the progression of renal fibrosis. This not only reduces the occurrence of Hypertension, but makes vasodilatation factor increase with the decrease of angiotensin. Besides blood circulation on cyst wall can be improved and blood vessels become flexible as well. What’s more, the active ingredients can target the damaged fibrosis tissues, Myofibroblast and immune complex. After the active ingredient reaches the targets, a range of effective components can be released which can degrade fibrous tissue and immune complex in the lesion. In addition, the active ingredient can supply requisite material to repair the damaged nephrons. Only renal structure gets repaired, can renal functions be rebuilt and recovered and clinical symptoms get relieved.